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Individuals harbor vastly different communities and abundances of myrosinase-producing bacteria (e.g., certain strains of Bacteroides, Enterococcus, and Lactobacillus), leading to profound differences in the efficiency of SFN generation ( 3.3 Host genetics: GST polymorphisms and metabolic destiny Beyond microbial activation, host genetics, particularly polymorphisms in GST genes, play a decisive role in shaping SFNs metabolic fate and tissue retention

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The vicious cycle of oxidative stress and carnitine dysregulation Persistent oxidative stress represents another central mechanism that exacerbates carnitine dysregulation in DKD [130,131,132,133]

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