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Together, these transcriptomic and functional data exclude a requirement for canonical MT1 or MT2 receptor signaling in melatonin-mediated regulation of ILC2 function

The underlying mechanism may involve the activation of the IGF pathway and its downstream signaling by downregulating IGFBP-7 expression, thereby promoting cellular proliferation

showed that ARID1A-deficient cancer cells expressed low levels of the key cystine transporter SLC7A11 and thus exhibited low basal levels of GSH, which made these cancer cells specifically vulnerable to inhibition of the GSH metabolic pathway 25

A randomised single blinded controlled trial