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We assume the value of E Cl to be such that Cl concentration is at equilibrium, that is the Cl synaptic influx and extrusion through KCC2 cancel each other out, this yields where V mean is the time averaged value the membrane potential taken to be V mean = 60 mV

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By contrast, if Roberts concept applies necrotizing agent ( i.e

This bidirectionality BPC-157 can both activate NO production in ischemic/hypoxic tissue and normalize NO dysregulation in inflammatory conditions suggests that the compound does not simply "increase" or "decrease" NO