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In the context of neurodegenerative diseases, the brains high oxygen consumption, abundant lipid content, and relatively low antioxidant capacity make it particularly vulnerable to oxidative stress [25,26,27]

Because new blood vessels underpin almost every repair process, this vascular action plausibly contributes to its effects in nerve, spinal cord, and brain models but it is a general repair mechanism, not a nerve-specific one

Although the overall disease progression of NAFLD has been characterized, the exact mechanism(s) underlying its initiation and progression remain poorly defined