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When there is excessive accumulation of LIP in chondrocytes, the overloaded Fe 2+ catalyzes the explosive generation of ROS through the iron-dependent Fenton reaction (Yang et al., 2024)

Their advantage lies in the ability to sensitively capture the early cascade of events, ranging from impaired mitochondrial fatty acid oxidation to transport defects and tubular metabolic imbalance, thereby complementing the molecular trajectory of DKD that is not adequately reflected by conventional UACR measurements (Table 1)

During the in vitro maturation of oocytes from aging mice, HSYCR upregulated SIRT3 expression, a key protein regulating mitochondrial function