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These two ghrelin receptor complexes display pronounced outward displacements of TM6 cytoplasmic end (~8 , measured at C of H258 6.30 ), the hallmark of GPCR activation, and ~3 inward shift of cytoplasmic end of TM7 (measured at C of Y323 7.53 ) (Fig

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This complex will then phosphorylate IB, making it susceptible to degradation by proteasomes, resulting in nuclear translocation of NF-B, which in turn initiates the transcription of many genes that promote inflammation