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In 1986, a group of doctors wrote in an article: This group (of 41 chronically ill patients) had somatization disorder diagnosed by specific criteria

As emphasized by Zhao et al., the prolonged progression of AD before clinical symptoms emerge indicates that astrocytes may substantially contribute to disease pathogenesis at a single-cell resolution 12

Recent advances in genetics and genomics have allowed for the identification of an increasingly high number of ASD-associated gene variants, including chromosomal deletions or duplications (copy number variations, CNVs), de novo and familial mutations, and epigenetic in candidate as well as novel genes [8,9,10]

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