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Wellness treatments focus on targeted symptom support rather than hormone optimization or regenerative signaling

The major problem to eliminate SnCs is their intrinsic resistance to apoptotic stimuli making most cytotoxic drugs inefficient 6,7

Oxidative stress also activates the Kelch-like ECH-associated protein 1 (Keap1)-Nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) redox regulator pathway, releasing Nrf2 to regulate the expression of genes that control antioxidant, inflammatory and immune system responses, facilitating GSH activity