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Given that numbers of inflammation-related diseases, such as rheumatoid arthritis, are associated with the downregulation of antioxidases 3,4 , while the direct administration of these natural antioxidases in pathological sites suffers from their over-large sizes leading to short circulating half-life, limited cell permeability, and antigenicity 5

doi: 10.1073/pnas.0813361106

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