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345,346 FOXO4-DRI, a cell-permeable peptide, disrupted the FOXO4-p53 interaction, thereby reducing senescence-induced FOXO4 activity and selectively targeting SnCs for p53-dependent apoptosis (Table 1)

Beta-blockers (BBs) are among the drugs that prevent the effects of endogenous catecholamine on -adrenergic receptors

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446 These inhibitors, such as geldanamycin, bind to the N-terminal nucleotide-binding pocket of HSP90 with higher affinity than ATP, leading to the proteasomal degradation of HSP90-dependent client proteins