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We propose that the GHK-Cu complex may act therapeutically against age-associated neurodegeneration and cognitive decline

The compound crosses the blood-brain barrier efficiently after oral administration and demonstrates sustained cognitive benefits even after treatment cessation, suggesting true structural enhancement rather than temporary stimulation

This Peptide is designed to stimulate lipolysis signaling while minimizing direct effects on insulin-like growth factor pathways in research settings

In the co-culture system, MCECs derived from the model group exhibited significant reduced ZO-1 and Occludin expression compared to those from the Control group, whereas GHK-Cu elevated theses protein levels (Figures 4GI)