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Ophthalmic Plast Reconstructive Surg (2021) 37(5):47681

This type of study is motivated by strong evidence that the moderate hyperbilirubinemia that occurs in individuals with Gilberts syndrome correlates with increased antioxidant status and decreased levels of pro-oxidant and pro-inflammatory markers

Instead of relying on standard mineral transport channels in the intestineswhich are often compromised in patients with chronic inflammation or gut dysbiosisthe chelated compound is absorbed further down the gastrointestinal tract via specialized dipeptide transport pathways

During postprandial periods there is an increase in free circulating IGF-1 via insulin-induced suppression of IGFBP-1 secretion [168170], which sequesters free IGF-1 making it unavailable