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The peptide activates extracellular signal-regulated kinase 1/2 (ERK1/2) phosphorylation in a dose-dependent manner, leading to nuclear translocation and activation of transcription factors including c-Fos, c-Jun, and early growth response-1 (Egr-1) that drive expression of genes involved in cell growth, migration, and angiogenesis

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Another significant advantage is its impact on metabolic health

By selectively inhibiting NNMT activity, researchers study how changes in nicotinamide metabolism influence intracellular NAD availability, methylation balance, mitochondrial efficiency, and lipid metabolism