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The Analyst 138 , 2840, (2013)

Third, because high-affinity sodium-dependent glutamate transport is impaired secondary to energy depletion and the resulting disturbance of ionic gradients (see Chapter 13 ), the energy depletion associated with bilirubin mitochondrial toxicity (see later) adds to the transport failure (or even reversal of transport) and accumulation of extracellular glutamate