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have tried to explain the better results of the nocturnal melatonin application by an additive effect of the endogenous and exogenous melatonin concentrations (Rateb et al., 2017

Nevertheless, technical complexity, ethical considerations, and potential immune responses remain major obstacles to clinical translation

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R389Q mouse model, suggesting that inflammation could be a major component of the pathology in the neurodegeneration of FDXR mutations [86] NDUFV1 deficiency (OMIM # 618225) NADH: ubiquinone oxireductase flavoprotein 1 (NDUFV1) is a 51-kDa highly conserved nuclear encoded subunit comprising the electron input (N) functional module of Complex I [87] )