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However, it can cause kidney damage, leading to impaired renal function, increased BUN and SCr levels, reduced glomerular filtration, and histopathological changes like mesangial cell hypercellularity, glomerular atrophy, tubular necrosis, degeneration, and vacuolization 7,8

It functions as a soluble decoy receptor comprising the extracellular domain of the activin type IIB receptor (ActRIIB) fused to an IgG1-Fc region

A noteworthy true history regarding this condition dates back to the 19th century when it was first identified by Sir James Paget , an English surgeon