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Finally, they reflect pathways likely additional to those involved in the angiogenesis (Vukojevic et al., 2018), recently associated in particular with the increased expression, internalization of VEGFR2, and the activation of the VEGFR2-Akt-eNOS signaling pathway ( Genes Expression as Support ), given the rapid presentation of all of these effects, which occurred before the process of angiogenesis could be initiated (Sikiric et al., 1993
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Taken together, the mechanisms position BPC-157 as a plausible agent for conditions where the rate-limiting steps in healing are vascularization, matrix remodeling and resolution of chronic low-grade inflammation
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