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Intracellular and extracellular redox status and free radical generation in primary immune cells from children with autism

In our recent studies, we have identified a different inhibition mechanism of TGR in which an inhibitor binds non-covalently at the so-called doorstop pocket interfering with enzyme function 33,34

Another goal of the study is to discover if this malfunction is related to an orexinergic deficiency and whether orexin-A may be used to restore it

The conversion of superoxide anions into hydrogen peroxide, before subsequently metabolized into water by catalase and glutathione (GSH) peroxidase, is facilitated by the metalloprotein superoxide dismutase (SOD)