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Found endogenously in human plasma (~200 ng/mL at age 20), saliva, and urine, GHK-Cu forms a high-affinity complex with copper(II) ions through its histidine imidazole nitrogen and peptide backbone coordination

In addition, the level of A42 was increased by miR-98 overexpression in the cell lysates of AD cell culture model (N2a/APP) while IGF-1 supplementation rescued the A42 accumulation, suggesting that miR-98 is involved in AD pathogenesis by negatively regulating IGF-1

Does not interfere with insulin sensitivity, making it potentially safer for individuals at risk of diabetes or metabolic disorders

Furthermore, the peptide component enhances cellular communication, signaling melanocytes to reduce overproduction of melanin while simultaneously promoting the repair of damaged skin structures that may perpetuate pigmentation problems